Submitted by Diane Hoffman on Sun, 09/13/2015

Alzheimer’s disease could be controlled with Resveratrol supplements: Research

A new research has indicated that a biomarker for Alzheimer’s disease can be impacted by intake of Resveratrol, a compound found in dark chocolate, red grapes and red wine. As Resveratrol can stabilize amyloid-beta40 levels in the blood and cerebrospinal fluid, it can reduce the formation of plaque in the brain.

Resveratrol is a compound found in abundance in red grapes, dark chocolate and red wine. It has been earlier credited for offering many health benefits and this is the reason, health studies conducted in the past have suggested eating dark chocolate and drinking moderate amount of red wine as a healthy option. A study conducted by medical researchers at Georgetown University has found that Resveratrol also affects an Alzheimer’s disease biomarker.

The research team found that amyloid-beta40 levels in the blood and cerebrospinal fluid remained stable among individuals who were given Resveratrol in their diet. For control group which was not given Resveratrol, the level of amyloid-beta40 witnessed a decline.

The research team conducted a double-blind, placebo controlled study. The study group included 119 participants, all with mild-to-moderate Alzheimer’s. The group given Resveratrol had mean age of 70 years while the placebo group had mean age of 73 years.

Amyloid-beta levels in Alzheimer’s patients decrease in the cerebrospinal fluid and deposit in the brain. These deposits become insoluble and lead to plaque formation, considered as a major reason for debilitating condition. The insoluble plaque ultimately leads to death of the nerve cells in the brain.

The research team added that additional studies will be needed before they could recommend people to increase their intake of dark chocolate and red wine.

The study has been published in Friday edition of the journal Neurology. The research team suggests that Resveratrol activates proteins called sirtuins. These proteins are also activated by calorie restriction, and may have anti-aging effects.

“It’s showing us a new mechanism, or a new pathway, towards Alzheimer’s treatments,” lead researcher Dr. R. Scott Turner, director of the Memory Disorders Program at Georgetown University Medical Center in Washington, D.C. said. “This is targeting amyloid in an indirect way,” Dr. Turner said.

The research team indicated that the Resveratrol given to the study subjects was equivalent of Resveratrol found in 10,000 bottles of red wine. This indicates that there will be a little positive impact of drinking red wine but as little as 50mg of pure red grape Resveratrol in supplement form would have the same effect and be a practical method of taking the required amount.

Good Life Labs Resveratrol

Hypertension, heart disease and blocked arteries, are the number one cause of deferment or denial of a pilot medical.

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These statements have not been evaluated by the FDA

This product is not intended to diagnose or cure any disease.

EDTA Removes Plaque from arteries

With a result that will surprise many (but not us), the largest trial of EDTA Chelation Therapy was undertaken by the National Institute of Health

The test followed 1,708 patients with coronary disease. A major benefit was found to be the substantially reduced need for surgical intervention to relieve blocked arteries. This was true to an even greater extent in patients who also had diabetes.

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Good Life Labs Customer Mrs M. from Gaffney, SC. remembers the day her husband visited the doctor for chronic heart disease. “They gave him six months to live”.

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Another article (Anderson, Hubacek, Wyse, and Knudtson, 2003) looked for evidence to see whether or not EDTA dilates arteries, which would support the theory of its increasing nitric oxide production in the endothelial cells. 

This double-blind, randomized study investigated the impact of infusions of EDTA with vitamins and minerals versus placebo (vitamins and minerals only) on endothelium-dependent brachial artery flow-mediated vasodilation in subjects with coronary artery disease who were also being “optimally treated with proven therapies for atherosclerotic risk factors.”  The researchers found significant differences in flow-mediated vasodilation between the treatment and placebo groups.

Other research has yielded some information about the possible mechanisms of EDTA in the body.  A study published in Free Radical Biology and Medicine (Hininger et al., 2005) investigated whether the large doses of vitamin C (an antioxidant) typically administered with EDTA therapy might actually have a prooxidant effect. 

The researchers gave two different groups of subjects EDTA with and without 5 g vitamin C and found that indeed the biochemical measurements of free radicals were markedly increased soon after therapy in the vitamin C group compared to the group that did not receive vitamin C.  However, over the course of 16 treatments, the researchers found that despite the prooxidant activity directly following each treatment, the long-term effects were antioxidant.  They suggested that this therapy might be beneficial to patients with heart disease and diabetes because the long term effects involved protection against lipid peroxidation. 

They also noted that “[t]he effects of multiple sessions of chelation therapy, without added vitamin C during the chelation session, may lead to even more beneficial effects and need to be determined.”  While this study is far from conclusive, it indicates that a component of the typical chelation treatment is likely beneficial, and it suggests fairly specific further study to continue learning about the potential antioxidant effects of EDTA. 

Other theories of how EDTA works have also been put forward. Since EDTA can bind to many different metals, it can trade one metal for another for which it has a higher affinity as it travels through the body.  This process theoretically has the potential to move trace elements from areas of high concentration (which, according to the cited research, can form in diseased tissue) to other areas where the elements can be of benefit in facilitating healthy enzymatic activity. 

What is perhaps a more commonly understood function of EDTA, the binding and removal of heavy metals that can interfere with enzymatic processes (although in mainstream practice this treatment is only used for known or suspected cases of heavy metal poisoning.

Yet another explanation for cardiovascular improvements after treatment with EDTA is that the removal of heavy metals leads to increased production of nitric oxide in the endothelial cells, causing the arteries to relax and improving flow. 

These explanations mostly center on the cardiovascular effects of ETDA, but its proponents think it is effective on a wide range of conditions.  One website explains how EDTA can achieve such broad improvements by saying that once the arteries are opened and the blood flow reestablished, other cells “downstream” will no longer be deprived of nutrients or the opportunity to eliminate waste.  The healing of these cells then ameliorates a number of diseases.

Many theories have been put forward for consideration [to explain how EDTA Chelation works.]  One of the more common explanations is that EDTA binds to the calcium in arterial plaques and removes it, thus breaking up the plaque.

 Some people express concern over the binding of calcium, thinking that this process must also strip away calcium from the bones and teeth.  Proponents of the theory explain that the calcium is only removed from inappropriate locations such as the arteries.  Garry Gordon, M.D., D.O., adds that the binding of calcium is actually good for the bones.  Reduced serum calcium levels stimulate the parathyroid gland to secrete parathormone, which not only causes the restoration of appropriate calcium levels but also activates bone-strengthening osteoblasts, effectively reversing the course of osteoporosis.

Cranton, however, rejects the whole theory of removing calcium from plaques as outdated.  He favors explanations involving more recent research into the area of free radical pathology.  Cranton explains that free radicals can disrupt many other important molecules around them and have been implicated in the promotion of atherosclerosis, cancer, and the basic decline in human health with age.  He says that EDTA binds to and removes a particular form of iron that can catalyze the formation of free radicals.

EDTA Oral Chelating Formula promotion